Dry flowable, water dispersible granule, flowable, wettable powder in water soluble bags.
PREMIX PARTNERS:Carbendazim; Chlorothalonil; Thiophanate-methyl; Thiram
ECOTOXICOLOGY
Birds Acute oral LD50 for quail >2510 mg/kg. LC50 for quail >5620 mg/kg.Fish LC50 (96 h) for trout 22-32, guppies 32.5, bluegill sunfish 50 mg/l.Daphnia LC50 (48 h) 4.0 mg/l.Bees Not toxic to bees. LD50 >200 mg/bee.Worms Not toxic to earthworms.
ENVIRONMENTAL FATE
Animals In the hen, the major metabolic routes are epoxidation of the vinyl group, followed by hydration of the intermediate epoxide, and by hydrolytic cleavage of the heterocyclic ring (G. M. Dean et al., Proc. Br. Crop Prot. Conf. - Pests Dis., 1988, 2, 693-698). In rats, following oral administration, eliminated in approximately equal proportions in the urine and faeces, with the principal metabolite being N-(3,5-dichlorophenyl)-2-methyl-2,3,4-trihydroxybutanamide. Plants In plants, the primary metabolites are (1-carboxy-1-methyl)allyl 3,5-dichlorophenylcarbamate and N-(3,5-dichlorophenyl)-2-hydroxy-2-methyl-3-butenamide. Alkaline hydrolysis leads to loss of 3,5-dichloroaniline from vinclozolin and its metabolites. The metabolites exist as conjugates.Soil/Environment Metabolism occurs by loss of the vinyl group, cleavage of the 5-membered ring and eventual formation of 3,5-dichloroaniline. Koc 100-735. Soil degradation of vinclozolin takes place with half-lives of several weeks, and mainly leads to the formation of bound residues.
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